🔒 Closed May ask lang medyo kinakabahan ako e

[daiken]

so anong balita sayo?

 

[Cute-Piggy]

1. Overview of how ulcers develop Although people talk about “stomach ulcers”, most peptic ulcers are actually duodenal ulcers as they are located in the first part of the duodenum. When food leaves the stomach it is saturated with hydrochloric acid and it is forced around a C-shape curve of the duodenum.

It is at the beginning of this C-shape wall of the duodenum that there is the highest risk of developing a duodenal ulcer. Peptic ulcers can develop when the normal defense mechanisms of the mucosa lining are undermined.

This can be from the chronic use of anti-inflammatory medication for arthritis (NSAIDs). But it can also be from a bacterium called Helicobacter pylori, or for short: H. Pylori.

This bacterium has developed a remarkable ability to survive in the acidy milieu of the stomach and duodenum as it produces several enzymes, which enable it to neutralize the acid in its immediate micro surrounding. It also produces mucolytic enzymes, which are capable of breaking down the superficial layer of the gastric and duodenal wall so that acid can now do the rest and erode the wall of the mucosa. A peptic ulcer (or simply ulcer) is a defect of the mucosal layer.

The term “peptic ulcer” comes from a time when physicians thought that ulcers would come from a combination of acid and the enzyme pepsin, which would in combination lead to the ulceration of the esophagus, stomach wall or duodenal wall. As pointed out above, we now know that defense mechanisms also play a tremendous role, as does a high secretion of ACTH and cortisol in the case of stress ulcers, which leads to a further weakening of these defense mechanisms. NSAIDs used for arthritis lead to a weakening of the repair mechanisms in the mucosal wall and increase the acidity on a cellular level, which means that the medication is “ulcerogenic”(it can cause ulcers).

Ulcers come in different forms. With NSAIDs it is often a multitude of erosions. These can be seen by gastroduodenoscopy. They are often located in the stomach, are very shallow and measure a few mm in diameter. Then there is a chronic duodenal ulcer, which may measure from 0.5 to 2 cm in diameter, and where the bacterium H. pylori may play a role as a chronic propagator. It is clear that any defect of the mucosa, which is full of blood vessels, can lead to bleeding ulcers. 2. Answering your question It is true that if a patient with an ulcer does not eat regularly the acid in the stomach will corrode the stomach further when there is no food to digest in the stomach for a long time. Doctors usually recommend the patient to take 5 small meals; this will bind the acid in the stomach by absorbing it in the food until it is digested and leaves the stomach. In the duodenum the milieu is alkaline and the acid is neutralized. Next the doctor will recommend some medicine that will suppress acid formation and this will also help the body to heal. Triple antibiotics are often also recommended when it is proven that H. pylori is involved. A well-researched remedy for stomach ulcers is licorice root. Licorice however contains glycyrrhetinic acid, which can elevate blood pressure. So scientists developed deglycyrrhizinated licorice tablets (DGL for short), which is very effective in relieving stomach ulcers, but has no side effect of elevating blood pressure. It comes in chewable tablets 380 to 400 mg of DGL per tablet. According to Murray (Ref. 1) the growth of H. pylori, the bacterium that causes difficult to heal ulcers is inhibited by DGL. In head-to-head studies, which are cited by Ref. 1 DGL was more effective than Zantac, Tagamet or antacids. Two to three chewable DGL tablets taken between meals or 20 minutes before meals will heal ulcers within 8 to 16 weeks. The difference is that with DGL the ulcer is healed, while with standard medication described above it is only symptomatically suppressed, but will often flare up when the treatment is stopped. DGL stimulates the normal defense mechanisms of the stomach and duodenum, improves the protective substances that line the intestinal wall and improves blood supply to the intestinal lining. The result is that the lifespan of the intestinal cell is prolonged.

Ref.1: Michael T. Murray, N.D.: “What the drug companies won’t tell you and your doctor doesn’t know” – The alternative treatments that may change your life – and the prescriptions that could harm you. Atria Books (subsidiary of Simon & Schuster Inc.), 2009 (page 73).

source:quora

 

[jackfrost75]

go to a doktor mhirap magtipid baka lalo ka pa mapasama

 

[Jmrie_]

go to a doktor mhirap magtipid baka lalo ka pa mapasama

Okay na po.

 

[Gaurum]

Mas maganda paconsult na po kayo sa clinic or hospital, mahirap na baka may ibang ailments kayo na hindi nyo pala alam na meron na pala kayo kaya nagkakaganyan.

 

[Kabayan09]

Pa check mo po. Doctor na titingin ang pinaka makakapag assess nung kalalagayan niyo po.

 

[PHC_demohenyo]

wag ka kabahan,madagdagan ng nervous breakdown yang sakit mo

 

[Jmrie_]

wag ka kabahan,madagdagan ng nervous breakdown yang sakit mo

Ganun ba?

 

[Jmrie_]

Pa check mo po. Doctor na titingin ang pinaka makakapag assess nung kalalagayan niyo po.

Okay na ako

 

[PHC_demohenyo]

kabag lng yan,éùtot mo lng

 

[Jmrie_]

kabag lng yan,éùtot mo lng

Opo salamat

 

[SometimesBad]

Um sumasakit ng konti itong tummy ko sa left side ng konti
Ulcer na ba matatawag yun madalas late na ako kumain eh..

Magpapacheck up rin ako tomorrow..
Gusto ko lang mag ask if my idea kayo..

Up

 

[erick11]

Kleo normal ba pag ihi mo?